Archived | Autism Speaks \ Cure Autism Now \ NAAR Publications: 2004 | #AutisticHistory #NotAnAutisticHistory

Publications: 2004

In addition to leveraging their AS/CAN/NAAR-funded studies to earn larger autism research grants from the National Institutes of Health and other sources, researchers funded by AS/CAN/NAAR have experienced significant success publishing their studies in numerous scientific journals and books. These publications play a key role in enhancing the scientific community’s understanding of autism spectrum disorders and elevating the caliber of the science. 

Autism Speaks is proud to present this partial list of articles that have resulted from AS/CAN/NAAR-funded research from 1996 to 2007. The funded researchers are listed in bold

Select from the following links to review publications for that year:

2007 | 2006 | 2005 | 2004 | 2003 | 2002 | 2001 | 2000 | 1999 | 1997/96


2004 

Belmonte MK, Cook EH Jr, Anderson GM, Rubenstein JL, Greenough WT, Beckel-Mitchener A, Courchesne E, Boulanger LM, Powell SB, Levitt PR, Perry EK, Jiang YH, DeLorey TM, and Tierney E. (2004) Autism as a disorder of neural information processing: directions for research and targets for therapy. Mol Psychiatry, 9(7):646-63.

Belmonte MK, Allen G, Beckel-Mitchener A, Boulanger LM, Carper RA, and Webb SJ. (2004) Autism and abnormal development of brain connectivity. J Neurosci, 24(42):9228-31.

Bomba MD and Pang EW. (2004) Cortical auditory evoked potentials in autism: a review. Int J Psychophysiol, (3):161-9.

Buxbaum JD, Silverman J, Keddache M, Smith CJ, Hollander E, Ramoz N, and Reichert JG. (2004) Linkage analysis for autism in a subset families with obsessive-compulsive behaviors: Evidence for an autism susceptibility gene on chromosome 1 and further support for susceptibility genes on chromosome 6 and 19. Mol Psychiatry, 9(2):144-150.

Cardy JEO, Ferrai P, Flagg EJ, Roberts W and Roberts TPL. (2004) Prominence of M50 auditory evoked response overM100 in childhood and autism. NeuroReport – Auditory and Vesibular Systems, 15: 1-4.

Chauhan A, Chauhan V, Brown T, and Cohen I. (2004) Oxidative stress in autism: increased lipid peroxidation and reduced serum levels of ceruloplasmin and transferrin–the antioxidant proteins. Life Sci, 75(21):2539-49.

Chauhan V, Chauhan A, Cohen IL, Brown WT, Sheikh A. (2004) Alteration in amino-glycerophospholipids levels in the plasma of children with autism: a potential biochemical diagnostic marker. Life Sci, 74(13):1635-43.

Chih B, Afrida SK, Clark L, and ScheiffeleP. (2004) Disorder-associated mutations lead to functional inactivation of neuroligins. Human Molecular Genetics, 13(14): 1471–147.

Comoletti D, De Jaco A, Jennings LL, Flynn R, Gaietta G and Tsigelny I. (2004) The Arg451Cys-neuroligin-3 mutation associated with autism reveals a defect in protein processing. Journal of Neuroscience, 24(20):4889–4893

Conciatori M, Stodgell CJ, Hyman SL, O’Bara M, Militerni R, Bravaccio C, Trillo S, Montecchi F, Schneider C, Melmed R, Elia M, Crawford L, Spence SJ, Muscarella L, Guarnieri V, D’Agruma L, Quattrone A, Zelante L, Rabinowitz D, Pascucci T, Puglisi-Allegra S, Reichelt KL, Rodier PM, and Persico AM. (2004) Association between the HOXA1 A218G polymorphism and increased head circumference in patients with autism. Biol Psychiatry, 55(4):413-9. 

Conroy J, Meally E, Kearney G, Fitzgerald M, Gill M and Gallagher L. (2004) Serotonin Transporter Gene and Autism: A Haplotype Analysis In an Irish Autistic Population. Molecular Psychiatry, 9: 587-593. 

Eason DD, Cannon JP, Haire RN, Rast JP, Ostrov DA, Litman GW. (2004) Mechanisms of antigen receptor evolution. Semin Immunol, 16(4):215-26.

Eden G, Turkeltaub PE, Lynn FD, Verbalis A, Miranda M, and Garcan L. (2004) The Neural Basis of Hyperlexic Reading: An fMRI Case Study. Neuron, 41:11-25. 

Engineer ND, Percaccio CR, Pandya PK, Moucha R, Rathbun DL, Kilgard MP. (2004) Environmental enrichment improves response strength, threshold, selectivity, and latency of auditory cortex neurons. J Neurophysiol, 92(1):73-82.

Ferland RJ, Eyaid W, Collura RV, Tully LD, Hill RS, Al-Nouri D, Al-Rumayyan A, Topcu M, Gascon G, Bodell A, Shugart YY, Ruvolo M, and Walsh CA. (2004) Abnormal cerebellar development and axonal decussation due to mutations in AHI1in Joubert syndrome. Nat Genet, 36(9):1008-13. Epub 2004 Aug 22. Erratum in: Nat Genet. 36(10):1126.

Filipek PA, Juranek J, Nguyen M, Cummings C and Gargus JJ. (2004) Relative Carnitine Deficiency in Autism. Journal of Autism and Developmental Disorders, 34(6):615-23. 

Herbert MR. (2004) Neuroimaging in disorders of social and emotional functioning: what is the question? J Child Neurol, 19(10):772-84.

Hernandez Prada JA, Haire RN, Cannon JP, Litman GW, Ostrov DA. (2004) Crystallization and preliminary X-ray analysis of VCBP3 from Branchiostoma floridae. Acta Crystallogr D Biol Crystallogr, (11):2022-4.

Hossain E, Wong BK, and Simpson EM. (2004) The dark phase improves genetic discrimination for some high throughput mouse behavioral phenotyping. Genes, Brain and Behavior, 3: 167–177.

Huentelman MJ, Zubcevic J, Hernandez Prada JA, Xiao X, Dimitrov DS, Raizada MK, Ostrov DA. (2004) Structure-based discovery of a novel angiotensin-converting enzyme 2 inhibitor. Hypertension, 44(6):903-6.

Ikuko T, Kudo LC, London SE, Geschwind DH and White S. (2004) Parallel FoxP1and FoxP2Expression in Songbird and Human Brain Predicts Functional Interaction. The Journal of Neuroscience, 24(13):3152–3163.

Jiang YHSahoo T, Michaelis RC, Bercovich D, Bressler J, Kashork CD, Liu Q, Shaffer LG, Schroer RJ, Stockton DW, Spielman RS, Stevenson RE, Beaudet A. (2004) A mixed epigenetic/genetic model for oligogenic inheritance of autism with a limited role for UBE3A. Am J Med Genet A, 131(1):1-10.

Jiang YH, Bressler J, Beaudet AL. (2004) Epigenetics and human disease. Annu Rev Genomics Hum Genet, 5:479-510.

Kramer J, Cushing BS, Carter CS, Wu J, and Ottinger MA. (2004) Sex and species differences in plasma oxytocin using an enzyme immunoassay. Canadian Journal of Zoology, 82:1194-1200.

Long JM, LaPorte P, Paylor R, Wynshaw-Boris A. (2004) Expanded characterization of the social interaction abnormalities in mice lacking Dvl1. Genes Brain Behav, 3(1):51-62.

Martin LA, Escher T, Goldowitz D, Mittleman G. (2004) A relationship between cerebellar Purkinje cells and spatial working memory demonstrated in a lurcher/chimera mouse model system. Genes Brain Behav, 3(3):158-66.

Martin-Ruiz CM, Lee M, Perry RH, Baumann M, Court JA, Perry EK. (2004) Molecular analysis of nicotinic receptor expression in autism. Brain Res Mol Brain Res, 123(1-2):81-90.

Massaro DW. (2004) Symbiotic Value of an Embodied Agent in Language Learning. In Proceedings of 37th Annual Hawaii International Conference on System Sciences, (HICCS’04)(CD-ROM,10 pages), Los Alimitos, CA: IEEE Computer Society Press.

Miller LJ and Hepburn S. (2004) An update on sensory responsivity, sensory reactivity, and arousal in persons with autism spectrum disorders: current research and promising interventions. Autism Asperger’s Digest, 38-40.

Mostofsky SH, Bunoski R, Morton SM, Goldberg M, and Bastian AJ. (2004) Children with Autism Adapt Normally during a Catching Task Requiring the Cerebellum. Neurocase, 10(1): 60–64.

Mukaetova-Ladinska EB, Arnold H, Jaros E, Perry R, and Perry E. (2004) Depletion of MAP2 expression and laminar cytoarchitectonic changes in dorsolateral prefrontal cortex in adult autistic individuals. Neuropathol Appl Neurobiol, 30(6):615-23.

Murcia CL, Bilovocky NA, and Herrup K. (2004) Dissecting complex genetic interactions that influence the Engrailed-1 limb phenotype Mammalian Genome,Vol. 15:352-360.

Nabi R, Serajee FJ, Chugani DC, Zhong H, and Huq AH. (2004) Association of tryptophan 2,3 dioxygenase gene polymorphism with autism. Am J Med Genet B Neuropsychiatr Genet, 125(1):63-8.

Ohtani N, Goto T, Waeber C, and Bhide PG. (2004) Dopamine modulates cell cycle in the lateral ganglionic eminence. Journal of Neuroscience, 23(7):2840-2850.

Patterson PH. (2004) Maternal infection causes altered behavior in the offspring. In “Neuropsychiatric Disorders and Infection,” 83-90. (Taylor & Francis)

Peden-Adams M, EuDaly J, Heesemann L, Smythe J, and Keil D. (2004) Postnatal Exposure to Thimerosal Alters Immunological Function in Adult Mice. The Toxicologist (Supplement to Toxicological Science), 2004 78 (S-1): 474.

Perrotti L, Hadeishi Y, Ulery P, Barrot M, Monteggia L, Duman R, and Nestler E. (2004) Induction of FosB in Reward-Related Brain Structures after Chronic Stress. Journal of Neuroscience, 24:10594-10602.

Perry EK and Perry RH. (2004) Neurochemistry of consciousness: cholinergic pathologies in the human brain. Prog Brain Res, 145:287-99.

Ramoz N, Reichert JG, Smith CJ, Silverman JM, Bespalova IN, Davis KL, and Buxbaum JD. (2004) Linkage and Association of the Mitochondrial Aspartate/Glutamate Carrier SLC25A12 Gene With Autism. Am J Psychiatry, 161(4):662-9.

Roberts T, Flagg EJ and Gage NM. (2004) Vowel categorization induces departure of M100 latency from acoustic prediction. NeuroReport – Cognitive Neuroscience and Neuropsychology, 19 (10): 1679-1682.

Serajee FJ, Nabi R, Zhong H, and Huq M. (2004) Polymorphisms in xenobiotic metabolism genes and autism. J Child Neurol, 19(6):413-7.

Takarae Y, Minshew NJ, Luna B, Krisky CM, and Sweeney JA. (2004) Pursuit eye movement deficits in autism. Brain, 127:2584-2594

Takarae Y, Minshew NJ, Luna B, and Sweeney JA. (2004) Oculomotor abnormalities parallel cerebellar histopathology in autism. Journal of Neurology, Neurosurgery, and Psychiatry, 75: 1359-1361. 

Theoret H, Kobayashi M, Merabet L, Wagner T, Tormos JM, and Pascual-Leone A. (2004) Modulation of right motor cortex excitability without awareness following presentation of masked self-images. Cognitive Brain Research, 20: 54-57.

Vincent JB, Thevarkunnel S, Kolozsvari D, Paterson AD, Roberts W, Scherer SW. (2004) Association and transmission analysis of the FMR1 IVS10 + 14C-T variant in autism. Am J Med Genet B Neuropsychiatr Genet, 125(1):54-6.

Williams JH, Massaro DW, Peel NJ, Bosseler A, Suddendorf T. (2004) Visual-auditory integration during speech imitation in autism. Res Dev Disabil, 25(6):559-75.

Xu J, Zwaigenbaum L, Szatmari P, and Scherer SW. (2004) Molecular Cytogentics of Autism. Current Genomics, 5: 347-364.

Yoder JA, Litman RT, Mueller MG, Desai S, Dobrinski KP, Montgomery JS, Buzzeo MP, Ota T, Amemiya CT, Trede NS, Wei S, Djeu JY, Humphray S, Jekosch K, Hernandez Prada JA, Ostrov DA, and Litman GW. (2004) Resolution of the novel immune-type receptor gene cluster in zebrafish. Proc Natl Acad Sci USA, 101(44):15706-11. Epub 2004 Oct 20.



Note/Warning:

Autistic people have fought the inclusion of ABA in therapy for us since before Autism Speaks, and other non-Autistic-led autism organizations, started lobbying legislation to get it covered by insurances and Medicaid. 

ABA is a myth originally sold to parents that it would keep their Autistic child out of an institution. Today, parents are told that with early intervention therapy their child will either be less Autistic or no longer Autistic by elementary school, and can be mainstreamed in typical education classes. ABA is very expensive to pay out of pocket. Essentially, Autism Speaks has justified the big price tag up front will offset the overall burden on resources for an Autistic’s lifetime. The recommendation for this therapy is 40 hours a week for children and toddlers.

The original study that showed the success rate of ABA to be at 50% has never been replicated. In fact, the study of ABA by United States Department of Defense was denounced as a failure. Not just once, but multiple times. Simply stated: ABA doesn’t workIn study after repeated study: ABA (conversion therapy) doesn’t work. 

What more recent studies do show: Autistics who experienced ABA therapy are at high risk to develop PTSD and other lifelong trauma-related conditions. Historically, the autism organizations promoting ABA as a cure or solution have silenced Autistic advocates’ opposition. ABA is also known as gay conversion therapy.


The ‘cure’ for Autistics not born yet is the prevention of birth. 

The ‘cure’ is a choice to terminate a pregnancy based on ‘autism risk.’ The cure is abortion. This is the same ‘cure’ society has for Down Syndrome. 

This is eugenics 2021. Instead of killing Autistics and disabled children in gas chambers or ‘mercy killings’ like in Aktion T4, it’ll happen at the doctor’s office, quietly, one Autistic baby at a time. Different approaches yes, but still eugenics and the extinction of an entire minority group of people.


Fact: You can’t cure Autistics from being Autistic.

Fact: You can’t recover an Autistic from being Autistic.

Fact: You can groom an Autistic to mask and hide their traits. Somewhat. … however, this comes at the expense of the Autistic child, promotes Autistic Burnout (this should not be confused with typical burnout, Autistic Burnout can kill Autistics), and places the Autistic child at high risk for PTSD and other lifelong trauma-related conditions.


[Note: Autism is NOT a disease, but a neurodevelopmental difference and disability.]


Fact: Vaccines Do Not Cause Autism.


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