In addition to leveraging their AS/CAN/NAAR-funded studies to earn larger autism research grants from the National Institutes of Health and other sources, researchers funded by AS/CAN/NAAR have experienced significant success publishing their studies in numerous scientific journals and books. These publications play a key role in enhancing the scientific community’s understanding of autism spectrum disorders and elevating the caliber of the science.
Autism Speaks is proud to present this partial list of articles that have resulted from AS/CAN/NAAR-funded research from 1996 to 2007. The funded researchers are listed in bold.
Androutsellis-Theotokis A, Goldberg NR, Ueda K, Beppu T, Beckman ML, Das S, Javitch JA, and Rudnick G. (2003) Characterization of a functional bacterial homologue of sodium-dependent neurotransmitter transporters. J Biol Chem, 278(15):12703-9.
Ang ES Jr, Haydar TF, Gluncic V, Rakic P. (2003) Four-dimensional migratory coordinates of GABAergic interneurons in the developing mouse cortex. J Biol Chem, 23(13):5805-15.
Belmonte MK and Yurgelun-Todd DA. (2003) Functional anatomy of impaired selective attention and compensatory processing in autism. Brain Res Cogn Brain Res, 17(3):651-64.
Bosseler A and Massaro DW. (2003) Development and evaluation of a computer-animated tutor for vocabulary and language learning in children with autism. J Autism Dev Disord, 33(6):653-72.
Cisternas FA, Vincent JB, Scherer SW, and Ray PN. (2003) Cloning and characterization of human CADPS and CADPS2, new members of the Ca2+-dependent activator for secretion protein family. Genomics, 81(3):279-91.
Cuccaro ML, Bass MP, Abramson RK, Ravan SA, Wright HH, Wolpert CM, Donnelly SL, and Pericak-Vance MA. (2003) Behavioral Comparisons in Autistic Individuals from Multilex and Singleton Families. Journal of Autism and Developmental Disabilities, 33(1):87-91.
DeSouza JF, Menon RS, and Everling S. (2003) Preparatory set associated with pro-saccades and anti-saccades in humans investigated with event-related FMRI. J Neurophysiol, 89(2):1016-23.
Ferland RJ, Cherry TJ, Preware PO, Morrisey EE, and Walsh CA. (2003) Characterization of Foxp2 and Foxp1 mRNA and protein in the developing and mature brain. J Comp Neurol, 460(2):266-79.
Grigorenko EL, Klin A, and Volkmar F. (2003) Annotation: Hyperlexia: disability or superability? J Child Psychol Psychiatry, 44(8):1079-91.
Haydar TF, Ang E Jr, Rakic P. (2003) Mitotic spindle rotation and mode of cell division in the developing telencephalon. Proc Natl Acad Sci USA, 100(5):2890-5.
Lugli G, Krueger JM, Davis JM, Persico AM, Keller F, and Smalheiser NR. (2003) Methodological factors influencing measurement and processing of plasma reelin in humans. BMC Biochemistry, 4:9.
Martin LA, Goldowitz D, Mittleman G. (2003) The cerebellum and spatial ability: dissection of motor and cognitive components with a mouse model system. Eur J Neurosci, 18(7):2002-10.
Massaro DW. (2003) A computer-animated tutor for spoken and written language learning. In “Proceedings of the 5th International Conference on Multimodal Interfaces” (ICMI’03) (pp.172-175). Vancouver, British Columbia, Canada. New York: ACM Press.
Massaro DW, Williams J, Suddendorf T, Bosseler A, and Peel N. (2003). Lip-reading and visual-auditory integration during speech imitation in autism. In “Proceedings of the 2nd International Symposium on Imitation in Animals and Artifacts” (AISB’03). Aberystwyth, U.K.
Massaro DW and Bosseler A. (2003) Perceiving Speech by Ear and Eye: Multimodal Integration by Children with Autism. The Journal of Developmental and Learning Disorders, 7: 111-146.
Nabi R, Zhong H, Serajee FJ, Huq AH. (2003) No association between single nucleotide polymorphisms in DLX6 and Piccolo genes at 7q21-q22 and autism. Am J Med Genet B Neuropsychiatr Genet, 119(1):98-101.
Newschaffer CJ and Curran LK. (2003) Autism: an emerging public health problem. Public Health Rep, 118(5):393-9.
Quattrocchi CC, Huang C, Niu S, Sheldon M, Benhayon D, Cartwright J, Mosier DR, Keller F, and D’Arcangelo G. (2003) Reelin promotes peripheral synapse elimination and maturation. Science, 301(5633):649-53.
Serajee FJ, Nabi R, Zhong H, and Mahbubul Huq AH. (2003) Association of INPP1, PIK3CG, and TSC2 gene variants with autistic disorder: implications for phosphatidylinositol signalling in autism. J Med Genet, 40(11):e119.
Serajee FJ, Zhong H, Nabi R, and Huq AH. (2003) The metabotropic glutamate receptor 8 gene at 7q31: partial duplication and possible association with autism. J Med Genet, 40(4):e42.
Shao Y, Cuccaro ML, Hauser ER, Raiford KL, Menold MM, Wolpert CM, Ravan SA, Elston L, Decena K, Donnelly SL, Abramson RK, Wright HH, DeLong GR, Gilbert JR, and Pericak-Vance MA. (2003) Fine mapping of autistic disorder to chromosome 15q11-q13 by use of phenotypic subtypes. American Journal of Human Genetics, 2(3):539-48.
Stone VE, Baron-Cohen S, Calder A, Keane J, and Young A. (2003) Acquired theory of mind impairments in individuals with bilateral amygdala lesions. Neuropsychologia, 41(2):209-20.
Takahashi T, Svoboda K, and Malinow R. (2003) Experience strengthening transmission by driving AMPA receptors into synapses. Science, 299(5612):1585-8.
Watson LR, Baranek GT, and DiLavore PC. (2003) Toddlers with Autism: Developmental Perspectives. Infants and Young Children, 16(3), pp.201-214.
Westphal V, Xiao M, Kwok PY, and Freeze HH. (2003) Identification of a frequent variant in ALG6, the cause of Congenital Disorder of Glycosylation-Ic. Hum Mutat, 22(5):420-1.
Zhong H, Serajee FJ, Nabi R, and Huq AH. (2003) No association between the EN2 gene and autistic disorder. J Med Genet, 40(1):e4.
Autistic people have fought the inclusion of ABA in therapy for us since before Autism Speaks, and other non-Autistic-led autism organizations, started lobbying legislation to get it covered by insurances and Medicaid.
ABA is a myth originally sold to parents that it would keep their Autistic child out of an institution. Today, parents are told that with early intervention therapy their child will either be less Autistic or no longer Autistic by elementary school, and can be mainstreamed in typical education classes. ABA is very expensive to pay out of pocket. Essentially, Autism Speaks has justified the big price tag up front will offset the overall burden on resources for an Autistic’s lifetime. The recommendation for this therapy is 40 hours a week for children and toddlers.
The original study that showed the success rate of ABA to be at 50% has never been replicated. In fact, the study of ABA by United States Department of Defense was denounced as a failure. Not just once, but multiple times. Simply stated: ABA doesn’t work. In study after repeated study: ABA (conversion therapy) doesn’t work.
What more recent studies do show: Autistics who experienced ABA therapy are at high risk to develop PTSD and other lifelong trauma-related conditions. Historically, the autism organizations promoting ABA as a cure or solution have silenced Autistic advocates’ opposition. ABA is also known as gay conversion therapy.
The ‘cure’ for Autistics not born yet is the prevention of birth.
The ‘cure’ is a choice to terminate a pregnancy based on ‘autism risk.’ The cure is abortion. This is the same ‘cure’ society has for Down Syndrome.
This is eugenics 2021. Instead of killing Autistics and disabled children in gas chambers or ‘mercy killings’ like in Aktion T4, it’ll happen at the doctor’s office, quietly, one Autistic baby at a time. Different approaches yes, but still eugenics and the extinction of an entire minority group of people.
Fact: You can’t cure Autistics from being Autistic.
Fact: You can’t recover an Autistic from being Autistic.
Fact: You can groom an Autistic to mask and hide their traits. Somewhat. … however, this comes at the expense of the Autistic child, promotes Autistic Burnout (this should not be confused with typical burnout, Autistic Burnout can kill Autistics), and places the Autistic child at high risk for PTSD and other lifelong trauma-related conditions.
[Note: Autism is NOT a disease, but a neurodevelopmental difference and disability.]
Fact: Vaccines Do Not Cause Autism.