Archived | Autism Speaks \ Cure Autism Now \ NAAR Publications: 2002 | #AutisticHistory #NotAnAutisticAlly

Publications: 2002

In addition to leveraging their AS/CAN/NAAR-funded studies to earn larger autism research grants from the National Institutes of Health and other sources, researchers funded by AS/CAN/NAAR have experienced significant success publishing their studies in numerous scientific journals and books. These publications play a key role in enhancing the scientific community’s understanding of autism spectrum disorders and elevating the caliber of the science. 

Autism Speaks is proud to present this partial list of articles that have resulted from AS/CAN/NAAR-funded research from 1996 to 2007. The funded researchers are listed in bold

Select from the following links to review publications for that year:

2007 | 2006 | 2005 | 2004 | 2003 | 2002 | 2001 | 2000 | 1999 | 1997/96


2002 

Alarcon M, Cantor RM, Liu J, Gilliam TC, and Geschwind DH. (2002) Evidence for a language quantitative trait locus on chromosome 7q in multiplex autism families. Am J Hum Genet, 70(1):60-71.

Betancur C,Corbex M, Spielewoy C, Philippe A, Laplanche JL, Launay JM, Gillberg C, Mouren-Simeoni MC, Hamon M, Giros B, Nosten-Bertrand M, and Leboyer M. (2002) Serotonin transporter gene polymorphisms and hyperserotonemia in autistic disorder. Mol Psychiatry, 7(1):67-71.

Blakely RD and Belous AR. (2002) Searching for novel genetic variation in neurotransmitter transporters. In “Transmembrane Transport: Receptor Biochemistry and Methodology Series.” D.R. Sibley and M.W. Quick (Eds.), 2002(5)65-87.

Buxbaum JD, Silverman JM, Smith CJ, Greenberg DA, Kilifarski M, Reichert J, Cook EH Jr, Fang Y, Song CY, and Vitale R. (2002) Association between a GABRB3 polymorphism and autism. Mol Psychiatry, 7(3):311-6.

Corbo JC, Deuel TA, Long JM, LaPorte P, Tsai E, Wynshaw-Boris A, Walsh CA. (2002) Doublecortin is required in mice for lamination of the hippocampus but not the neocortex. J Neurosci, 22(17):7548-57.

Freeze HH. (2002) Human disorders in N-glycosylation and animal models. Biochim Biophys Acta, 1573(3):388-93.

Gregory C, Lough S, Stone V, Erzinclioglu S, Martin L, Baron-Cohen S, and Hodges JR. (2002) Theory of mind in patients with frontal variant frontotemporal dementia and Alzheimer’s disease: theoretical and practical implications. Brain, 125(Pt 4):752-64.

Herzing LB, Cook EH Jr, and Ledbetter DH. (2002) Allele-specific expression analysis by RNA-FISH demonstrates preferential maternal expression of UBE3A and imprint maintenance within 15q11- q13 duplications. Hum Mol Genet, 11(15):1707-18.

Inoue S, Shimoda M, Nishinokubi I, Siomi MC, Okamura M, Nakamura A, Kobayashi S, Ishida N, and Siomi H. (2002) A role for the Drosophila fragile X-related gene in circadian output. Curr Biol, 12(15):1331-5.

Jamain S, Betancur C, Quach H, Philippe A, Fellous M, Giros B, Gillberg C, Leboyer M, and Bourgeron T. (2002) Linkage and association of the glutamate receptor 6 gene with autism. Mol Psychiatry, 7(3):302-10.

Kim SJ, Herzing LB, Veenstra-VanderWeele J, Lord C, Courchesne R, Leventhal BL, Ledbetter DH, Courchesne E, and Cook EH Jr. (2002) Mutation screening and transmission disequilibrium study of ATP10C in autism. Am J Med Genet, 114(2):137-43.

Klin A, Jones W, Schultz RVolkmar F,and Cohen S. (2002) Defining and Quantifying the Social Phenotype in Autism. American Journal of Psychiatry, Vol. 159: 895-908.

Lauritsen MB, Borglum AD, Betancur C, Philippe A, Kruse TA, Leboyer M, and Ewald H. (2002) Investigation of two variants in the DOPA decarboxylase gene in patients with autism. Am J Med Genet , 114(4):466-70.

Lee M, Martin-Ruiz C, Graham A, Court J, Jaros E, Perry R, Iversen P, Bauman M, and Perry E. (2002) Nicotinic receptor abnormalities in the cerebellar cortex in autism. Brain, 125(Pt 7):1483-95.

Madsen KM, Hvid A, Vestergaard M, Schendel D, Wohlfahrt J, Thorsen P, Olsen J, and Melbye M. (2002) A Population-Based Study of Measles, Mumps, and Rubella Vaccination and Autism. New England Journal of Medicine, 347:1477-1482.

Rinehart NJ, Bradshaw JL, Tonge BJ, Brereton AV, and Bellegrove MA. (2002) A neurobehavioural examination of individuals with high-functioning autism and Asperger’s disorder using a fronto-striatal model of dysfunction. Behavioural and Cognitive Neuroscience Reviews, 2002:1(2), 164-177.

Rinehart NJ, Bradshaw JL, Brereton AV, and Tonge BJ. (2002) Lateralization in individuals with high-functioning autism and Asperger’s disorder: a frontostriatal model. Aust N Z J Psychiatry, 36(6):762-70.

Rinehart NJ, Bradshaw JL, Brereton AV, and Tonge BJ. (2002) Lateralization in individuals with high-functioning autism and Asperger’s disorder: a frontostriatal model. J Autism Dev Disord, 32(4):321-331.

Romanski LM, Goldman-Rakic PS. (2002) An auditory domain in primate prefrontal cortex. , Nat Neurosci., 5(1):15-6.

Shumyatsky GP, Tsvetkov E, Malleret G, Vronskaya S, Hatton M, Hampton L, Battey JF, Dulac C, Kandel ER, and Bolshakov VY. (2002) Identification of a signaling network in lateral nucleus of amygdala important for inhibiting memory specifically related to learned fear. Cell, 111(6):905-18.

Siomi MC, Higashijima K, Ishizuka A, and Siomi H. (2002) Casein kinase II phosphorylates the fragile X mental retardation protein and modulates its biological properties. Mol Cell Biol, 22(24):8438-47.

Sparks BF, Friedman SD, Shaw DW, Aylward EH, Echelard D, Artru AA, Maravilla KR, Giedd JN, Munson J, Dawson G, and Dager SR. (2002) Brain structural abnormalities in young children with autism spectrum disorder. Neurology, 59(2):184-92.

Torres AR, Maciulis A, Stubbs EG, Cutler A, and Odell D. (2002) The transmission disequilibrium test suggests that HLA-DR4 and DR13 are linked to autism spectrum disorder. Hum Immunol, 63(4):311-6.

Vincent JB, Petek E, Thevarkunnel S, Kolozsvari D, Cheung J, Patel M, and Scherer SW. (2002) The RAY1/ST7 tumor-suppressor locus on chromosome 7q31 represents a complex multi-transcript system. Genomics, 80(3):283-94.

Williams MA, Moss SA, Bradshaw JL, and Rinehart NJ. (2002) Random number generation in autism. J Autism Dev Disord, 32(1):43-7.



Note/Warning:

Autistic people have fought the inclusion of ABA in therapy for us since before Autism Speaks, and other non-Autistic-led autism organizations, started lobbying legislation to get it covered by insurances and Medicaid. 

ABA is a myth originally sold to parents that it would keep their Autistic child out of an institution. Today, parents are told that with early intervention therapy their child will either be less Autistic or no longer Autistic by elementary school, and can be mainstreamed in typical education classes. ABA is very expensive to pay out of pocket. Essentially, Autism Speaks has justified the big price tag up front will offset the overall burden on resources for an Autistic’s lifetime. The recommendation for this therapy is 40 hours a week for children and toddlers.

The original study that showed the success rate of ABA to be at 50% has never been replicated. In fact, the study of ABA by United States Department of Defense was denounced as a failure. Not just once, but multiple times. Simply stated: ABA doesn’t workIn study after repeated study: ABA (conversion therapy) doesn’t work. 

What more recent studies do show: Autistics who experienced ABA therapy are at high risk to develop PTSD and other lifelong trauma-related conditions. Historically, the autism organizations promoting ABA as a cure or solution have silenced Autistic advocates’ opposition. ABA is also known as gay conversion therapy.


The ‘cure’ for Autistics not born yet is the prevention of birth. 

The ‘cure’ is a choice to terminate a pregnancy based on ‘autism risk.’ The cure is abortion. This is the same ‘cure’ society has for Down Syndrome. 

This is eugenics 2021. Instead of killing Autistics and disabled children in gas chambers or ‘mercy killings’ like in Aktion T4, it’ll happen at the doctor’s office, quietly, one Autistic baby at a time. Different approaches yes, but still eugenics and the extinction of an entire minority group of people.


Fact: You can’t cure Autistics from being Autistic.

Fact: You can’t recover an Autistic from being Autistic.

Fact: You can groom an Autistic to mask and hide their traits. Somewhat. … however, this comes at the expense of the Autistic child, promotes Autistic Burnout (this should not be confused with typical burnout, Autistic Burnout can kill Autistics), and places the Autistic child at high risk for PTSD and other lifelong trauma-related conditions.


[Note: Autism is NOT a disease, but a neurodevelopmental difference and disability.]


Fact: Vaccines Do Not Cause Autism.


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