In addition to leveraging their AS/CAN/NAAR-funded studies to earn larger autism research grants from the National Institutes of Health and other sources, researchers funded by AS/CAN/NAAR have experienced significant success publishing their studies in numerous scientific journals and books. These publications play a key role in enhancing the scientific community’s understanding of autism spectrum disorders and elevating the caliber of the science.
Autism Speaks is proud to present this partial list of articles that have resulted from AS/CAN/NAAR-funded research from 1996 to 2007. The funded researchers are listed in bold.
Belmonte M and Yurgelun-Todd D. (2001) Permutation Testing Made Practical for Functional Magnetic Resonance Image Analysis. IEEE Transactionals on Medical Imaging, 20(3): 243-248.
Blatt GJ, Fitzgerald CM, Guptill JT, Booker AB, Kemper, TL and Bauman, ML. (2001) Density and Distribution of Hippocampal Neurontransmitter Receptors in Autism: An autoradiographic study. Journal of Autism and Developmental Disorders, 31(6): 537-544.
Buchsbaum MS, Hollander E, Haznedar MM, Tang C, Spiegel-Cohen J, Wei TC, Solimando A, Buchsbaum BR, Robins D, Bienstock C, Cartwright C, and Mosovich S. (2001) Effect of fluoxetine on regional cerebral metabolism in autistic spectrum disorders: a pilot study. Int J Neuropsychopharmacol, 4(2):119-25.
Buxbaum JD, Silverman JM, Smith CJ, Kilifarski M, Reichert J, Hollander E, Lawlor BA, Fitzgerald M, Greenberg DA, and Davis KL. (2001) Evidence for a susceptibility gene for autism on chromosome 2 and for genetic heterogeneity. Am J Hum Genet, 68(6):1514-2.
Cheung J, Petek E, Nakabayashi K, Tsui LC, Vincent JB, and Scherer SW. (2001) Identification of the human cortactin-binding protein-2 gene from the autism candidate region at 7q31. Genomics, 78(1-2):7-11.
Ferguson JN, Aldag JM, Insel TR and Young LJ. (2001) Oxytocin in the Medical Amygdala is Essential for Social Recognition in the Mouse. Journal of Neuroscience, 21(20):8278-8285.
Folstein S and Sheidley B. (2001) Genetics of Autism: Complex Aetiology for a Heterogeneous Disorder. Nature Reviews Genetics, 943-955.
Fonseca MI, Ni YG, Dunning DD, and Miledi R. (2001) Distribution of Serotonin 2A, 2C and 3 Receptor mRNA in Spinal Cord and Medulla Oblongata Brain Research Molecular Brain Res, 18,89 (1-2): 11-9.
Freeze HH and Westphal V. (2001) Balancing N-linked glycosylation to avoid disease. Biochimie, 83(8):791-9.
Freeze HH. (2001) Update and perspectives on congenital disorders of glycosylation. Glycobiology, 11(12):129R-143R.
Hemby S, Sanchez MM, and Winslow JT. (2001) Gene Expression Profiling of Autism: Alterations in Temporal Lobe Profiles. Journal of Autism and Developmental Disorders, 31(6): 551-556.
Herzing LB, Kim SJ, Cook EH Jr, and Ledbetter DH. (2001) The human aminophospholipid-transporting ATPase gene ATP10C maps adjacent to UBE3A and exhibits similar imprinted expression. Am J Hum Genet, 68(6):1501-5.
Hollander E, Dolgoff-Kaspar R, Cartwright C, Rawitt R, and Novotny S. (2001) An Open Trial of Divalproex Sodium in Autism Spectrum Disorders. Journal of Clinical Psychiatry, 62(7):530-4.
Junaid MA and Pullarkat RK. (2001) Proteomic Approach for the Elucidation of Biological Defects in Autism. Journal of Autism and Developmental Disorders, 31(6): 557-560.
Liu J, Nyholt DR, Magnussen P, Parano E, Pavone P, Geschwind D, Lord C, Iversen P, Hoh J, Ott J, Gilliam TC. (2001) A genomewide screen for autism susceptibility loci. Am J Hum Genet, 69(2):327-40.
Ni YG, Chen JG, Androutsellis-Theotokis A, Haung CJ, Moczydlowski E and Rudnick G. (2001) A Lithium-Induced Conformational Change in Serotonin Transporter Alters Cocaine Binding, Ion Conductance, and Reactivity of Cys-109. Journal of Biology and Chemistry, 276(33):30942-30947.
Perry EK, Lee ML, Martin-Ruiz CM, Court JA, Volsen SG, Merrit J, Folly E, Iversen PE, Bauman ML, Perry RH, and Wenk GL. (2001) Cholinergic activity in autism: abnormalities in the cerebral cortex and basal forebrain. Am J Psychiatry, 158(7):1058-66.
Perry EK, Martin-Ruiz CM, and Court JA. (2001) Nicotinic receptor subtypes in human brain related to aging and dementia, Alcohol, 24(2):63-8.
Persico AM, D’Agruma L, Maiorano N, Totaro A, Militerni R, Bravaccio C, Wassink TH, Schneider C, Melmed R, Trillo S, Montecchi F, Palermo M, Pascucci T, Puglisi-Allegra S, Reichelt K-L, Conciatori M, Marino R, Baldi A, Zelante L, Gasparini P, and Keller F. (2001) Reelin Gene Alleles and Haplotypes as a Factor Predisposing to Autistic Disorder. Molecular Psychiatry, 6(2): 152-159.
Petek E, Windpassinger C, Vincent JB, Cheung J, Boright AP, Scherer SW, Kroisel PM, and Wagner K. (2001) Disruption of a novel gene (IMMP2L) by a breakpoint in 7q31 associated with Tourette syndrome. Am J Hum Genet, 68(4):848-58.
Purcell AE, Rocco MM, Lenhart JA, Hyder K, Zimmerman AW, and Pevsner J. (2001) Assessment of Neural Cell Adhesion Molecule (NCAM) in Autistic Serum and Postmortem Brain. Journal of Autism and Developmental Disorders, 2:183-94.
Rinehart NJ, Bradshaw JL, Brereton AV, and Tonge BJ. (2001) Movement preparation in high-functioning autism and Asperger disorder: a serial choice reaction time task involving motor reprogramming. J Autism Dev Disord, 31(1):79-88.
Rinehart NJ, Bradshaw JL, Moss SA, Brereton AV, Tonge BJ. (2001) A deficit in shifting attention present in high-functioning autism but not Asperger’s. Autism, 5(1):67-80.
Schumann CM, Buonocore MH, and Amaral DG. (2001) Magnetic Resonance Imaging of the Post-Mortem Autistic Brain. Journal of Autism and Developmental Disorders, 31(6): 561-568.
Shavelle RM, Strauss DJ, and Pickett J. (2001) Causes of Death in Autism. Journal of Autism and Developmental Disorders, 31(6): 569-576.
Stodgell CJ, Hyman SL, and Ingram JL. (2001) The Role of Candidate Genes in Unraveling the Genetics of Autism. International Review of Research in Mental Retardation, 23: 57-81.
Weidenheim KM. (2001) Neuropatologia del Autismo. Neurociencia, 2(3): 133-137.
Weidenheim KM, Goodman L, Dickson DW, Gillberg C, Rastam M, and Rapin I. (2001) Etiology and Pathophysiology of Autistic Behavior: Clues From Two Cases With an Unusual Variant of Neuroaxonal Dystrophy. Journal of Child Neurology, 16(11):809-819.
Whitaker-Azmitia PM. (2001) Serotonin and Brain Development: Role in Human Developmental Diseases. Brain Research Bulletin, 56(5):479-85.
Young LJ, Pitkow LJ, and Ferguson JN. (2001) Neuropeptides and Social Behavior: Animal Models Relevant to Autism. Molecular Psychiatry, 7 Suppl 2:S38-9.
Autistic people have fought the inclusion of ABA in therapy for us since before Autism Speaks, and other non-Autistic-led autism organizations, started lobbying legislation to get it covered by insurances and Medicaid.
ABA is a myth originally sold to parents that it would keep their Autistic child out of an institution. Today, parents are told that with early intervention therapy their child will either be less Autistic or no longer Autistic by elementary school, and can be mainstreamed in typical education classes. ABA is very expensive to pay out of pocket. Essentially, Autism Speaks has justified the big price tag up front will offset the overall burden on resources for an Autistic’s lifetime. The recommendation for this therapy is 40 hours a week for children and toddlers.
The original study that showed the success rate of ABA to be at 50% has never been replicated. In fact, the study of ABA by United States Department of Defense was denounced as a failure. Not just once, but multiple times. Simply stated: ABA doesn’t work. In study after repeated study: ABA (conversion therapy) doesn’t work.
What more recent studies do show: Autistics who experienced ABA therapy are at high risk to develop PTSD and other lifelong trauma-related conditions. Historically, the autism organizations promoting ABA as a cure or solution have silenced Autistic advocates’ opposition. ABA is also known as gay conversion therapy.
The ‘cure’ for Autistics not born yet is the prevention of birth.
The ‘cure’ is a choice to terminate a pregnancy based on ‘autism risk.’ The cure is abortion. This is the same ‘cure’ society has for Down Syndrome.
This is eugenics 2021. Instead of killing Autistics and disabled children in gas chambers or ‘mercy killings’ like in Aktion T4, it’ll happen at the doctor’s office, quietly, one Autistic baby at a time. Different approaches yes, but still eugenics and the extinction of an entire minority group of people.
Fact: You can’t cure Autistics from being Autistic.
Fact: You can’t recover an Autistic from being Autistic.
Fact: You can groom an Autistic to mask and hide their traits. Somewhat. … however, this comes at the expense of the Autistic child, promotes Autistic Burnout (this should not be confused with typical burnout, Autistic Burnout can kill Autistics), and places the Autistic child at high risk for PTSD and other lifelong trauma-related conditions.
[Note: Autism is NOT a disease, but a neurodevelopmental difference and disability.]
Fact: Vaccines Do Not Cause Autism.