In addition to leveraging their AS/CAN/NAAR-funded studies to earn larger autism research grants from the National Institutes of Health and other sources, researchers funded by AS/CAN/NAAR have experienced significant success publishing their studies in numerous scientific journals and books. These publications play a key role in enhancing the scientific community’s understanding of autism spectrum disorders and elevating the caliber of the science.
Autism Speaks is proud to present this partial list of articles that have resulted from AS/CAN/NAAR-funded research from 1996 to 2007. The funded researchers are listed in bold.
Baranek GT. (2000) The early identification of infants with autism. DD Research Digest, North Carolina Developmental Disabilities Research Consortium, Winter, 8(3): 20-22.
Bass MP, Menold MM, Wolpert CM, Donnelly SL, Ravan SA, Hauser ER, Maddox LO, Vance JM, Abramson RK, Wright HH, Gilbert JR, Cuccaro ML, DeLong GR, and Pericak-Vance MA. (2000) Genetic Studies in Autistic Disorder and Chromosome 15. Neurogenetics, 2(4): 219-26.
Donnelly SL, Wolpert CM, Menold MM, Bass MP, Gilbert JR, Cuccaro ML, Delong GR, and Pericak-Vance MA. (2000) Female with Autistic Disorder and Monosomy X (Turner Syndrome): Parent-of Origin Effect of the X Chromosome American. Journal of Medical Genetics, 96(3): 312-6.
Everling S. (2000) Neuronal correlates for preparatory set associated with pro-saccades and anti-saccades in the primate frontal eye field. J Neurosci, 20(1):387-400.
Ferguson JN, Young LJ, Hearn EF, Matzuk MM, Insel TR and Winslow JT. (2000) Social Amnesia in Mice Lacking the Oxytocin Gene. Nature Genetics, 25(3): 284-8.
Goldberg MC, Landa R, Lasker A, Cooper L, and Zee DS (2000) Evidence of Normal Cerebellar Control of the Vestibulo-Ocular Reflex (VOR) in Children with High-Functioning Autism. Journal of Autism and Developmental Disorders, 30(6): 519-24.
Haznedar MM, Buchsbaum MS, Wei T, Hof PR, Cartwright C, Bienstock CA, and Hollander E. (2000) Circuit in Autism Spectrum Illnesses Studied with Positron Emission Tomography and Magnetic Resonance Imaging. American Journal of Psychiatry, 157(12): 1994-2001.
Hollander E, Novotny S, Allen A, Aronowitz B, Cartwright C, and DeCaria C. (2000) The Relationship Between Repetitive Behaviors and Growth Hormone Response to Sumatriptan Challenge in Adult Autistic Disorder. Neuropsychopharmacology, 22(2): 163-7.
Hong SE, Shugart YY, Huang DT, Shahwan SA, Grant PE, O’B. Hourihane J, Martin NDT, and Walsh CA. (2000) Autosomal Recessive Lissencephaly with Cerebellar Hypoplasia is Associated with Human RELN Mutations. Nature Genetics, 26:93-6.
Hurley RA, Lewine JD, Jones GM, Orrison WW Jr, and Taber KH. (2000) Application of magnetoencephalography to the study of autism. J Neuropsychiatry Clin Neurosci, 12(1):1-3.
Ingram JL, Stodgell CJ, Hyman SL, Figlewicz DA, Weitkamp LR, and Rodier PM. (2000) Discovery of Allelic Variants of HOXA1 and HOXB1: Genetic Susceptibility to Autism Spectrum Disorders. Teratology, 62(6): 393-405.
Lin PT, Gleeson JG, Corbo JC, Flanagan L, and Walsh CA. (2000) DCAMKL1 Encodes a Protein Kinase with Homology to Doublecortin that Regulates Microtubal Polymerization. The Journal of Neuroscience,Vol. 20(24):9152-9161.
Martin ER, Menold MM, Wolpert CM, Bass M, Donnelly SL, Ravan SA, Zimmerman A, Gilbert JR, Vance JM, Maddox LO, Wright HH, Abramson RK, DeLong GR, Cuccaro ML, and Pericak-Vance MA. (2000) Analysis of Linkage Disequilibrium in Gamma-Aminobutyric Acid Receptor Subunit Genes in Autistic Disorder. American Journal of Medical Genetics, 96(1): 43-8.
Messer A and Kang XW. (2000) Control of transcription in the RORa-Staggerer Mutant Mouse Cerebellum: Glutamate Receptor Delta2 mRNA. International Journal of Developmental Neuroscience, 18(7):663-8.
Novotny S, Hollander E, Allen A, Mosovich S, Aronowitz B, Cartwright C, DeCaria C, and Dolgoff-Kaspar R. (2000) Increased Growth Hormone Response to Sumatriptan Challenge in Adult Autistic Disorders. Psychiatry Research, 94(2): 173-177.
Rinehart NJ, Bradshaw JL, Moss SA, Brereton AV, and Tonge BJ. (2000) Atypical interference of local detail on global processing in high-functioning autism and Asperger’s disorder. J Child Psychol Psychiatry, 41(6):769-78.
Sandler RH, Finegold SM, Bolte ER, Buchanan CP, Maxwell AP, Vaisanen ML, Nelson MN, Wexler HM. (2000) Short-term benefit from oral vancomycin treatment of regressive-onset autism. J Child Neurol, 15(7):429-35.
Schultz RT, Romanski LM, and Tsatsanis KD. (2000) Neurofunctional Model of Autistic Disorder and Asperger Syndrome. In “Asperger Syndrome”, A. Klin, F.R. Volkmar and S.S. Sparrow, Eds. (Guilford Press: New York).
Smith M, Filipek PA, Wu C, Bocian HS, Modahl C, and Spence MA. (2000) Analysis of a 1-Megabase Deletion in 15q22-q23 in an Autistic Patient: Identification of Candidate Genes for Autism and Homologous DNA Segments in 15q22-q23 and 15q11-q13. American Journal of Medical Genetics, 96(6): 765-70.
Stone VE. (2000) The role of the frontal lobes and the amygdala in theory of mind. In “Understanding Other Minds: Perspectives from Autism and Developmental Cognitive Neuroscience.” Chapter 11:pps.253-272.
Vincent JB, Herbrick JA, Gurling HM, Bolton PF, Roberts W, and Scherer SW. (2000) Identification of a novel gene on chromosome 7q31 that is interrupted by a translocation breakpoint in an autistic individual. Am J Hum Genet, 67(2):510-4.
Walsh CA and Goffinet AM. (2000) Potential Mechanisms of Mutations that Affect Neuronal Migration in Man and Mouse. Current Opinion in Genetics and Development, 10:270-274.
Whitmore TE, Holloway JL, Lofton-Day CE, Maurer MF, Chen L, Quinton TJ, Vincent JB, Scherer SW, and Lok S. (2000) Human secretin (SCT): gene structure, chromosome location, and distribution of mRNA. Cytogenet Cell Genet, 90(1-2):47-52.
Wolpert CM, Menold MM, Bass MP, Qumsiyeh MB, Donnelly SL, Ravan SA, Vance JM, Gilbert JR, Abramson RK, Wright HH, Cuccaro ML, and Pericak-Vance MA. (2000) Three Probands with Autistic Disorder and Isodicentric Chromosome 15. American Journal of Medical Genetics, 96(3): 365-72.
Autistic people have fought the inclusion of ABA in therapy for us since before Autism Speaks, and other non-Autistic-led autism organizations, started lobbying legislation to get it covered by insurances and Medicaid.
ABA is a myth originally sold to parents that it would keep their Autistic child out of an institution. Today, parents are told that with early intervention therapy their child will either be less Autistic or no longer Autistic by elementary school, and can be mainstreamed in typical education classes. ABA is very expensive to pay out of pocket. Essentially, Autism Speaks has justified the big price tag up front will offset the overall burden on resources for an Autistic’s lifetime. The recommendation for this therapy is 40 hours a week for children and toddlers.
The original study that showed the success rate of ABA to be at 50% has never been replicated. In fact, the study of ABA by United States Department of Defense was denounced as a failure. Not just once, but multiple times. Simply stated: ABA doesn’t work. In study after repeated study: ABA (conversion therapy) doesn’t work.
What more recent studies do show: Autistics who experienced ABA therapy are at high risk to develop PTSD and other lifelong trauma-related conditions. Historically, the autism organizations promoting ABA as a cure or solution have silenced Autistic advocates’ opposition. ABA is also known as gay conversion therapy.
The ‘cure’ for Autistics not born yet is the prevention of birth.
The ‘cure’ is a choice to terminate a pregnancy based on ‘autism risk.’ The cure is abortion. This is the same ‘cure’ society has for Down Syndrome.
This is eugenics 2021. Instead of killing Autistics and disabled children in gas chambers or ‘mercy killings’ like in Aktion T4, it’ll happen at the doctor’s office, quietly, one Autistic baby at a time. Different approaches yes, but still eugenics and the extinction of an entire minority group of people.
Fact: You can’t cure Autistics from being Autistic.
Fact: You can’t recover an Autistic from being Autistic.
Fact: You can groom an Autistic to mask and hide their traits. Somewhat. … however, this comes at the expense of the Autistic child, promotes Autistic Burnout (this should not be confused with typical burnout, Autistic Burnout can kill Autistics), and places the Autistic child at high risk for PTSD and other lifelong trauma-related conditions.
[Note: Autism is NOT a disease, but a neurodevelopmental difference and disability.]
Fact: Vaccines Do Not Cause Autism.